Some studies suggest that hyperbaric oxygen therapy (HBOT) contributes to tissue and blood vessel regeneration, potentially aiding in combating degeneration.
The therapy promotes the formation of new blood vessels in regions of the body with compromised circulation and is employed by certain physicians to assist in the treatment of Coronary Heart Disease, Macular Degeneration, Parkinson’s Disease, Alzheimer’s Disease, Arthritis, and immune-related disorders.
HBOT additionally aids in promoting collagen activation, combating signs of aging such as skin damage and loss of elasticity.
Clinical studies have illustrated the advantages of HBOT for age-related degenerative conditions, offering cellular support to all organs in the body, thereby promoting overall health and beauty.
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We’ve chosen several research articles to share with you regarding hyperbaric oxygen therapy and its role in supporting age-related conditions and anti-aging strategies.
BDNF, or Brain-Derived Neurotrophic Factor, is a protein that the body releases to stimulate the production of new brain cells through a process known as neurogenesis. This protein is crucial for supporting learning, memory, higher thinking, and other cognitive functions. Notably, reduced levels of BDNF have been observed in the brains of individuals with Alzheimer’s, Parkinson’s, and Huntington’s disease. Researchers are actively studying BDNF and its impact on brain plasticity and regeneration. It is well-established that hyperbaric oxygen therapy (HBOT) can promote brain regenerative effects. A recent study, published in 2017, further supports this notion. Researchers utilized both lower pressure (1.5 ATA) and higher pressure (2.0 ATA) HBOT sessions and found significant increases in BDNF levels after just 3 to 5 consecutive days of treatment.
“After five days of hyperbaric oxygen therapy (HBOT), both the genetically modified NIH3T3/BDNF and native NIH3T3 fibroblasts exhibited significantly increased proliferation compared to normoxic controls.”
A study published in 2012 in “Cell Stress and Chaperones, A Comprehensive Journal of Stress Biology and Medicine” investigated the effects of HBOT preconditioning and its protective effects against Ultraviolet-A (UV-A) induced skin damage. Hairless mice were divided into three groups and exposed to UV-A three days a week for 22 weeks. Two of the groups received HBOT pretreatment either two or four times a week. UV-A exposure increased skin cell death, indicating heightened levels of skin damage. However, pretreatment with HBOT significantly reduced UV-A induced cell death. Furthermore, HBOT pretreatment prevented skin creasing and maintained skin elasticity.
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